Filter-And-Forward Distributed Beamforming in Relay Networks with Frequency Selective Fading

A new approach to distributed cooperative beamforming in relay networks with frequency selective fading is proposed. It is assumed that all the relay nodes are equipped with finite impulse response (FIR) filters and use a filter-and-forward (FF) strategy to compensate for the transmitter-to-relay and relay-to-destination channels. Three relevant half-duplex distributed beamforming problems are considered. The first problem amounts to minimizing the total relay transmitted power subject to the destination quality-of-service (QoS) constraint. In the second and third problems, the destination QoS is maximized subject to the total and individual relay transmitted power constraints, respectively. For the first and second problems, closed-form solutions are obtained, whereas the third problem is solved using convex optimization. The latter convex optimization technique can be also directly extended to the case when the individual and total power constraints should be jointly taken into account. Simulation results demonstrate that in the frequency selective fading case, the proposed FF approach provides substantial performance improvements as compared to the commonly used amplify-and-forward (AF) relay beamforming strategy.Comment: Submitted to IEEE Trans. on Signal Processing on 8 July 200

The catalogues and mid-infrared environment of Interstellar OH Masers

Data for a number of OH maser lines have been collected from surveys. The posi- tions are compared to recent mid-infrared (MIR) surveys such as Spitzer-GLIMPSE and WISE, restricting the comparison to point sources. The colors and intensities of the IR sources are compared. There are many 18 cm OH masers, but far fewer in lines arising from higher energy levels. We also make a comparison with the 5 cm Class II methanol masers. We have divided the results into 3 subsamples: those associated with OH masers only, those associated with OH masers and Class II methanol masers, and those only associated with Class II methanol masers. There are no obvious dif- ferences in the color-color or color-magnitude results for the GLIMPSE point sources. However, according to the results from the WISE 22 {\mu}m survey, the sources associ- ated with OH masers are brighter than those associated with methanol masers. We interpret the presence of OH and methanol masers mark the locations of regions where stars are forming. The OH masers are located on the borders of sharp features found in the IR. These are referred to as bubbles. If the OH masers mark the positions of protostars, the result provides indirect evidence for triggered star formation caused by the expansion of the bubbles.Comment: 23 pages (11 pages online only), 12 figures, Accepted. Monthly Notices of the Royal Astronomical Society,201

A Prospective Study of the Incidence of Retinopathy of Prematurity in China: Evaluation of Different Screening Criteria

To investigate the incidence of Retinopathy of Prematurity (ROP) in Beijing, North China, and to evaluate the effectiveness of different ROP screening criteria, we conducted a prospective cohort study in a single-neonatal intensive care unit (NICU). A total of 2997 premature infants with birth weight (BW) ≤ 2000 g and/or gestational age (GA) ≤ 34 weeks had completed ROP screening. ROP was diagnosed in 356 (11.9%) infants. The mean GA was 30.46 ± 1.98 weeks and the mean BW was 1477.35 ± 371.29 g. Of the 59 (2.0%) infants receiving treatment, the mean GA was 29.37 ± 2.10 weeks, and the mean BW was 1240.80 ± 330.71 g. The incidence of ROP declined from 14.7% in 2009 and 11.1% in 2010 to 9.5% in 2011. The United Kingdom (UK) criteria could reduce the screening number by 40.8%, and 3 infants with type I ROP needing treatment were missed, but none in 2011. The United States (US) criteria could reduce the screening number by 66.5%, and 10 infants with type I ROP needing treatment were missed, including one in 2011. So the UK criteria may be appropriate for screening of ROP in our NICU in 2011. Future multisite epidemiologic studies are required to establish suitable ROP screening criteria in China

Sparse general non-negative matrix factorization based on left semi-tensor product

The dimension reduction of large scale high-dimensional data is a challenging task, especially the dimension reduction of face data and the accuracy increment of face recognition in the large scale face recognition system, which may cause large storage space and long recognition time. In order to further reduce the recognition time and the storage space in the large scale face recognition systems, on the basis of the general non-negative matrix factorization based on left semi-tensor (GNMFL) without dimension matching constraints proposed in our previous work, we propose a sparse GNMFL/L (SGNMFL/L) to decompose a large number of face data sets in the large scale face recognition systems, which makes the decomposed base matrix sparser and suppresses the decomposed coefficient matrix. Therefore, the dimension of the basis matrix and the coefficient matrix can be further reduced. Two sets of experiments are conducted to show the effectiveness of the proposed SGNMFL/L on two databases. The experiments are mainly designed to verify the effects of two hyper-parameters on the sparseness of basis matrix factorized by SGNMFL/L, compare the performance of the conventional NMF, sparse NMF (SNMF), GNMFL, and the proposed SGNMFL/L in terms of storage space and time efficiency, and compare their face recognition accuracies with different noises. Both the theoretical derivation and the experimental results show that the proposed SGNMF/L can effectively save the storage space and reduce the computation time while achieving high recognition accuracy and has strong robustness

GENERATING TRAINING DATABASES USED IN VECTOR BASED OBJECT RECOGNITION IN HYBRID CLOUD USING PUBLIC PROFILES

Techniques are provided herein for generating a face data set which contains badge identifier photos and photos from social media. The faces are automatically tagged using facial recognition, text recognition, and human relationship mining

Identity Authentication Security Management in Mobile Payment Systems

Mobile payment is a new payment method offering users mobility, reachability, compatibility, and convenience. But mobile payment involves great uncertainty and risk given its electronic and wireless nature. Therefore, biometric authentication has been adopted widely in mobile payment in recent years. However, although technology requirements for secure mobile payment have been met, standards and consistent requirements of user authentication in mobile payment are not available. The flow management of user authentication in mobile payment is still at its early stage. Accordingly, this paper proposes an anonymous authentication and management flow for mobile payment to support secure transaction to prevent the disclosure of users\u27 information and to reduce identity theft. The proposed management flow integrates transaction key generation, encryption and decryption, and matching to process users\u27 personal information and biometric characteristics based on mobile equipment authentication carrier

A rare novel mutation in TECTA causes autosomal dominant nonsyndromic hearing loss in a Mongolian family

BACKGROUND: The genetic basis of autosomal dominant nonsyndromic hearing loss is complex. Genetic factors are responsible for approximately 50% of cases with congenital hearing loss. However, no previous studies have documented the clinical phenotype and genetic basis of autosomal dominant nonsyndromic hearing loss in Mongolians. METHODS: In this study, we performed exon capture sequencing of a Mongolian family with hereditary hearing loss and identified a novel mutation in TECTA gene, which encodes α -tectorin, a major component of the inner ear extracellular matrix that contacts the specialized sensory hair cells. RESULTS: The novel G → T missense mutation at nucleotide 6016 results in a substitution of amino acid aspartate at 2006 with tyrosine (Asp2006Tyr) in a highly conserved zona pellucida (ZP) domain of α-tectorin. The mutation is not found in control subjects from the same family with normal hearing and a genotype-phenotype correlation is observed. CONCLUSION: A novel missense mutation c.6016 G > T (p.Asp2006Tyr) of TECTA gene is a characteristic TECTA-related mutation which causes autosomal dominant nonsyndromic hearing loss. Our result indicated that mutation in TECTA gene is responsible for the hearing loss in this Mongolian family

SUMOylation of Grb2 enhances the ERK activity by increasing its binding with Sos1

BACKGROUND: Grb2 (Growth factor receptor-bound protein 2) is a key adaptor protein in maintaining the ERK activity via linking Sos1 (Son of sevenless homolog 1) or other proteins to activated RTKs, such as EGFR. Currently, little knowledge is available concerning the post-translational modification (PTM) of Grb2 except for its phosphorylation. Since emerging evidences have highlighted the importance of SUMOylation (Small ubiquitin-related modifier), a reversible PTM, in modulating protein functions, we wondered if Grb2 could be SUMOylated and thereby influences its functions especially involved in the Ras/MEK/ERK pathway. METHODS: SUMOylation of Grb2 was analyzed with the in vivo SUMOylation assay using the Ni(2+)-NTA affinity pulldown and the in vitro E.coli-based SUMOylation assay. To test the ERK activity and cell transformation, the murine fibroblast cell line NIH/3T3 and the murine colon cancer cell line CMT-93 were used for the experiments including Grb2 knockdown, ectopic re-expression, cell transformation and migration. Immunoprecipitation (IP) was employed for seeking proteins that interact with SUMO modified Grb2. Xenograft tumor model in mice was conducted to verify that Grb2 SUMOylation regulated tumorigenesis in vivo. RESULTS: Grb2 can be SUMOylated by SUMO1 at lysine 56 (K(56)), which is located in the linker region between the N-terminal SH3 domain and the SH2 domain. Knockdown of Grb2 reduced the ERK activity and suppressed cell motility and tumorigenesis in vitro and in vivo, which were all rescued by stable ectopic re-expression of wild-type Grb2 but not the mutant Grb2(K56R). Furthermore, Grb2 SUMOylation at K(56) increased the formation of Grb2-Sos1 complex, which sequentially leads to the activation of Ras/MEK/MAPK pathway. CONCLUSIONS: Our results provide evidences that Grb2 is SUMOylated in vivo and this modification enhances ERK activities via increasing the formation of Grb2-Sos1 complex, and may consequently promote cell motility, transformation and tumorigenesis